Raymond Fucini
Mentor: Jeffrey Pessin, Ph.D.
Chen D, Fucini RV, Olson AL, Hemmings BA, Pessin JE. Osmotic shock inhibits insulin signaling by maintaining Akt/protein kinase B in an inactive dephosphorylated state. Mol Cell Biol. 1999 Jul;19(7):4684-94. PubMed PMID:10373517; PubMed Central PMCID: PMC84266.
Fucini RV, Okada S, Pessin JE. Insulin-induced desensitization of extracellular signal-regulated kinase activation results from an inhibition of Raf activity independent of Ras activation and dissociation of the Grb2-SOS complex. J Biol Chem. 1999 Jun 25;274(26):18651-8. PubMed PMID: 10373477.
Zhao H, Li YY, Fucini RV, Ross SE, Pessin JE, Koretzky GA. T cell receptor-induced phosphorylation of Sos requires activity of CD45, Lck, and protein kinase C, but not ERK. J Biol Chem. 1997 Aug 22;272(34):21625-34. PubMed PMID: 9261185.
Chen D, Fucini RV, Olson AL, Hemmings BA, Pessin JE. Osmotic shock inhibits insulin signaling by maintaining Akt/protein kinase B in an inactive dephosphorylated state. Mol Cell Biol. 1999 Jul;19(7):4684-94. PubMed PMID:10373517; PubMed Central PMCID: PMC84266.
Fucini RV, Okada S, Pessin JE. Insulin-induced desensitization of extracellular signal-regulated kinase activation results from an inhibition of Raf activity independent of Ras activation and dissociation of the Grb2-SOS complex. J Biol Chem. 1999 Jun 25;274(26):18651-8. PubMed PMID: 10373477.
Zhao H, Li YY, Fucini RV, Ross SE, Pessin JE, Koretzky GA. T cell receptor-induced phosphorylation of Sos requires activity of CD45, Lck, and protein kinase C, but not ERK. J Biol Chem. 1997 Aug 22;272(34):21625-34. PubMed PMID: 9261185.